Rheumatoid arthritis (RA) is a general inflammatory disease mostly featured by synovitis and joint destruction. In other words, it is an autoimmune disease that causes prolonged swelling of the joints and the tissue around the joints and other tissues. The etiology of rheumatoid arthritis is unknown. However, the effects of genetic features are apparent, the genetic foundation is insufficient to describe the initiation of the immune insult ( Woetzel et al., 2014) . The leading characteristic, in this case, is inflammation with the predominant characteristic being the synovium. osteoarthritis was originally thought of as a wear and tear infection that attacked individuals as they aged. The pathogenesis of osteoarthritis is characterized by deprivation of cartilage and restoration of underlying bone as a result of an effective reaction of chondrocytes in the inflammatory cells and the articular cartilage in the adjacent tissues. The discharge of enzymes from these cells decomposes proteoglycans and collagen, damaging the articular cartilage.
Osteoarthritis and rheumatoid arthritis (RA) are two different types of arthritis. They both share some comparable properties, although each has different symptoms and needs different treatment. As a result, an appropriate diagnosis is necessary for both. Osteoarthritis tends to be the most widespread type of arthritis. However, rheumatoid arthritis strikes nearly one-tenth as many individuals as osteoarthritis ( Kalkbrenner, Schmidt, & Penlesky, 2014) . Osteoarthritis comes about when the smooth cartilage joint tissue wears out. Osteoarthritis often starts in a remote joint. Rheumatoid arthritis is an autoimmune illness, which implies that the immune system fails and damages the body rather than the intruders.
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The major disparity between rheumatoid arthritis and osteoarthritis is the cause at the back of the joint symptoms. Rheumatoid arthritis is an autoimmune infection whereby the body’s individual immune system strikes the body joint tissues. It usually attacks minor and major joints on both sides of the body symmetrically, such as balls of both feet, both elbows, both hands and wrists ( Withrow et al., 2016) . Whereas osteoarthritis is caused by involuntary wear and tear on the joints. Signs usually start in one area of the body and may extend to the other regions. Symptoms start progressively and are normally restricted to a single set of joints, typically the finger joints next to the thumb or the fingernails, the spine or great weight-bearing joints including the hips and the knees.
Rheumatoid arthritis (RA) and osteoarthritis (OA) are two of the most widespread musculoskeletal disorders affecting individuals across the United States. Well-known by cartilage disintegration and bony overgrowth, OA affects nearly 13.9% of adults who are older than or at the age of 25 ( Andersen et al., 2014) . Although old age is the major risk factor for osteoarthritis, it is in any way an unavoidable consequence of getting old. Radiographic variations of osteoarthritis, especially osteophytes, are normal among the aged population but signs of joint pain may be liberated of radiographic impact in the majority of older adults.
Osteoarthritis is a progressive articular disease with intricate pathogeny since varied features interact instigating a course of progressive loss of the cartilage. Regardless of the complex nature of this pathology, since the 1950's it is recognized that particular types of osteoarthritis are linked to a solid genetic component ( Andersen et al., 2014) . The genomic bases of these infections do not keep to the characteristic arrangements of Mendelian heritage and perhaps they are associated with changes in various genes. The detection of a large number of aspirant genes to deliberate exposure to the growth of the osteoarthritis confirms the multifactorial nature of this illness.
Rheumatoid arthritis is an autoimmune disease that results in chronic swelling of the joints and the tissue around the joints and other tissues while osteoarthritis is a disease characterized by deprivation of cartilage and restoration of underlying bone as a result of an effective reaction of chondrocytes in the inflammatory cells and the articular cartilage in the adjacent tissues ( Wojdasiewicz, Poniatowski, & Szukiewicz, 2014) . The identification of a huge number of aspirant genes to deliberate exposure to the growth of the osteoarthritis and endorses the multifactorial nature of this illness. Reducing the inflammation and pain of both osteoarthritis and rheumatoid arthritis, a doctor is required to prescribe acetaminophen or a nonsteroidal anti-inflammatory drug (NSAID), such as Aleve ( Wojdasiewicz et al., 2014) . The doctor may as well recommend corticosteroid joint injections to reduce stiffness and the pain of affected joints.
References
Andersen, M., Ellegaard, K., Hebsgaard, J. B., Christensen, R., Torp-Pedersen, S., Kvist, P. H., & Danneskiold-Samsøe, B. (2014). Ultrasound colour Doppler is related to synovial pathology in operations from hand intersections in rheumatoid arthritis patients: a cross-sectional training. Registers of the Rheumatic Illnesses , 73 (4), 678-683.
Kalkbrenner, A. E., Schmidt, R. J., & Penlesky, A. C. (2014). Ecological biochemical experiences and autism spectrum conditions: a review of the epidemiological indication. Existing Problems in Pediatric and Adolescent Health Care , 44 (10), 277-318.
Withrow, J., Murphy, C., Liu, Y., Hunter, M., Fulzele, S., & Hamrick, M. W. (2016). Extracellular vesicles in the pathogenesis of rheumatoid arthritis and osteoarthritis. Arthritis Research & Therapy , 18 (1), 286.
Woetzel, D., Huber, R., Kupfer, P., Pohlers, D., Pfaff, M., Driesch, D., & Kinne, R. W. (2014). Association of rheumatoid arthritis and osteoarthritis patients by transcriptome-based rule set generation. Arthritis Study & Treatment , 16 (2), R84.
Wojdasiewicz, P., Poniatowski, Ł. A., & Szukiewicz, D. (2014). The character of inspiring and anti-inflammatory cytokines in the pathogenesis of osteoarthritis. Mediators of Inflammation , 2014 .
