Pollutant and pollutants which caused the most adverse health outcome are chemical toxins such the Polycyclic aromatic hydrocarbon (PAH), Organochlorines DDT, and PCBs (Meeker, 2012). Various adverse health outcomes occurred as a result of exposure to these chemical toxins. They include reproductive capacity being adversely affected regarding reduced fertility, congenital disabilities, children born with low birth weight as well as children's developmental disabilities. Also, exposure to these chemical toxins caused liver cancer in human beings (Covello & Merkhoher, 2013). To prevent adverse health outcomes associated with these chemical toxins from occurring some steps were suggested. One, pregnant women were advised to avoid exposure to Polycyclic aromatic hydrocarbon (PAH) which affects their reproductive capacity (Covello & Merkhoher, 2013). Secondly, workers were also advised to avoid direct exposure to PCBs as they increase their chances of getting liver cancer and malignant melanoma. Another step that is necessary to prevent these adverse health outcomes is to advise nursing mothers who are exposed to toxic chemicals at their workplace that they ought to stop nursing their babies as this would prevent chemical exposure routes for the infants through breast milk. Several measures may be undertaken to prevent future exposure to harmful chemical toxins. These will include substituting plastic for glass or metal which are more environmentally friendly, avoiding storage of certain chemicals in the house, and choosing foodstuffs wisely, especially fish which are often high in DDT and PCBs also the federal government should regulate the production of these toxic chemicals as they might cause adverse health outcomes (Meeker, 2012). There is a strong association between environmental hazards and adverse health outcomes in humans. This is because various environmental hazards such as air pollution especially Sulphur dioxide and other pollutants are strongly linked to causing various respiratory diseases such as asthma, bronchitis, and other lung diseases. Also, studies have revealed that exposure to toxic chemical pollutants especially PCBs increases the risk of disruption of reproductive functions (Govarts ET.AL 2013).
There are several exposure-disease relationships associated with chemical toxins Polycyclic aromatic hydrocarbon (PAH), Organochlorines DDT, and PCBs. First, Polychlorinated Biphenyls (PCBs) are associated with the disruption of reproductive functions and also cause developmental effects in kids among others. On the other hand, Polycyclic aromatic hydrocarbon (PAH) is associated with pulmonary, gastrointestinal, renal, and dermatologic systems diseases in humans (Govarts ET.AL 2013). Further, high-level exposure to some organochlorine residues is associated with causing abnormalities of liver function, skin as well as the nervous system.
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References
Covello, V. T., & Merkhoher, M. W. (2013). Risk assessment methods: approaches for assessing health and environmental risks . Springer Science & Business Media.
Govarts, E., Nieuwenhuijsen, M., Schoeters, G., Ballester, F., Bloemen, K., De Boer, M., ... & Legler, J. (2012). Birth weight and prenatal exposure to polychlorinated biphenyls (PCBs) and dichlorodiphenyldichloroethylene (DDE): a meta-analysis within 12 European Birth Cohorts. Environmental health perspectives , 120 (2), 162.
Meeker, J. D. (2012). Exposure to environmental endocrine disruptors and child development. Archives of pediatrics & adolescent medicine , 166 (10), 952-958.
