Cancer is the abnormal growth of cells. Most research has been focusing on establishing the cause of abnormal cell development. Some investigations have successfully determined the cause, while there is still no correlation with other factors such as family history. For instance, there is enough evidence that skin cancer can result from the sun's ultraviolet rays. However, there is not enough evidence on the cause of other types of cancer, such as prostate. There is a connection between family members through genes. In reproduction, the genes are transferred from the parents to the offspring, carrying certain traits. Some of the characteristics are genetic diseases like sickle cell anemia. But there is a need to know if it is possible to transmit cancer through a similar process. It will be possible to create preventive strategies once the route cause is determined. Family connections have been assessed as the leading cause of prostate cancer (Hemminki & Czene, 2002). However, a critical challenge is the similarity of individuals' lifestyles, making both parties affected. For instance, the father and the son have similar lifestyle arrangements, suggesting that they might suffer from similar ailments.
However, there is a lack of an assertion on how a father can transmit cancer to the son through the genetic makeup. There is a high recurrence rate among the makes who have brothers or fathers who have been prostate cancer victims (Zeegers, Jellema & Ostrer, 2003). Also, the disease's recurrence is more common in the first generation than in the second and third. There is a hereditary connection to the issue of prostate cancer among men. But there is a need to include the aspects of technological advancement. The risk of prostate cancer among men might have been high in the first generation due to inferior technology in detecting and managing the condition. The situation cannot be compared to the current era, where it is possible to see the growth early and implement intervention measures. Family medical history might have some contribution but not on more significant levels. It can just be considered a risk factor where if an individual has a family history of prostate cancer, they should consider going for regular check-ups. Assumptions cannot be made as the condition is life-threatening. Therefore, there is a need for more studies to establish the connection between hereditary genes and prostate cancer.
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The genes are carriers of many other diseases transmitted to the offspring. However, there is no clear indication that the chromosomes can equally be carriers of the prostate cancer strain. Studies indicate that those affected by prostate cancer have a second-degree relative that has been a victim (Fuchs et al., 1994). But it is not sufficient to conclude a correlation between the two. Some of the second-degree individuals are not even close relatives. Carriers of a disease can be seen in approximately the third the fourth generations. Lack of sufficient evidence on the hereditary transmission of the disease makes it easy to dwell more on an individual's lifestyle. The main focus is on food consumption that is a crucial contributor. The current generation's prostate cancer rate is much higher than in medieval times. The main change that has taken place is on the lifestyle. Individuals in the millennial generation mainly consume processes foods with high chemicals that are not good for the body.
References
Fuchs CS, et al. (1994). A prospective study of family history and the risk of colorectal cancer. N Engl J. Med. 331 (25), 69-74.
Hemminki K. & Czene K. (2002). Age-specific and attributable risks of familial prostate carcinoma from the family-cancer database . National Center for Biotechnology Information, 95 (6), 1346-53.
Zeegers M.P, Jellema A. & Ostrer H. (2003). Empiric risk of prostate carcinoma for relatives of patients with prostate carcinoma: a meta-analysis. ACS journals, National Center for Biotechnology Information , 97 (8),1894-903.
