The term nanobacteria comes from the fusion of the word ‘nano’ and ‘bacteria’. ‘Nano’ is the Latin term for extremely small. Thus, nanobacteria refers to extremely small bacterial organism whose size is approximately 0.01 the size of other bacteria and are claimed to have been discovered in living tissue and rock (Rona, 2005). Nanobacteria discharge an adhesive, calcium-rich crust that lets them to stick each other and to the cells inside blood vessel walls. The calcium coat then hardens into a shell, defending the microbes from the immune system as well as shielding them from radiation, all antibiotics, and also chemotherapy. As they remain concealed in the blood vessel walls, they trigger an inflammatory cascade in the blood vessel or organ that eventually forms a rigid calcific plaque. Over the years, the plaque layers progressively increase, thereby causing diseases in arteries or organs.
Owing to their minute size, nanobacteria traverse through the filters used conventionally and can find their way into vaccines and other biological cures, thereby contaminating them. As such, it requires application of special techniques to isolate and culture the nanobacteria. Notable techniques include the dark field microscopy and a number of blood culturing methods. These techniques have revealed the existence of various bacteria and other microorganisms in the bloodstreams of non-sick people. Currently, there are essentially two methods of determining if nanobacteria exist in an individual’s body. The first method involves running tests for nanobacteria through examining the individual’s blood sample in a well-equipped lab to conduct the calcium bomb test (Rona, 2005). However, the most accurate method involves conducting a blood test that isolates both antigens and antibodies associated to nanobacteria. A different method involves conducting a special kind of cardiac CT scan that computes the calcium score. The interpretation is that a higher calcium score indicates a higher likelihood of the individual to be suffering from a nanobacterial infection.
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This leads to an important question, ‘what infections or diseases are associated with nanobacteria?’ Notably, a host of diseases have been found to be closely linked with nanobacteria. These include bladder stones, breast implant calcification, kidney stones, aortic valve sclerosis, bone spurs, calcium deposits in the skin (calcinosis cutis), and prostatic stones (Rona, 2005). From various scientific investigations, it has been established that no nutritional intervention, medication, or therapy has presented reliable evidence of reversing every quantifiable indicator of heart disease. Various symptoms tested include clotting, inflammation, as well as hard and soft plaques that comprise of calcium deposits. The reason for this phenomenon is attributed to the effects of nanobacteria since they cause calcium deposits in protecting themselves from the immune system, radiation, and antibiotics.
With the knowledge of nanobacteria, efforts have been devoted towards developing treatments for the diseases associated with nanobacteria. The approach towards treating the diseases involves destroying the nanobacteria, consequently reversing the diseases they cause. The process deals with stripping them of their calcium crust using EDTA (ethylenediamine tetra-acetic acid) followed by doses of tetracycline to attack the suddenly uncovered nanobacteria (Rona, 2005). As of now, tetracycline remains the single antibiotic established to be effective against nanobacteria. In fact, recent experiments dealing with varying the typical intravenous chelation treatment showed it to work effectively in reversing diseases triggered by nanobacteria.
So, what is the EDTA chelation therapy? It is a process that utilizes a solution of amino acids which in turn dissolves any deposits of calcium present in the body. The main acknowledged medical procedure for applying the chelation therapy is in ridding the body of poisonous heavy metals such as aluminum, lead, mercury, copper, cadmium, arsenic, and other toxins. However, a number of doctors have applied chelation therapy in the treatment of various diseases such as atherosclerosis, Alzheimer’s disease, coronary artery disease, intermittent claudication (cramping pain and weakness in the legs), high blood pressure, angina, diabetes, macular degeneration, and other blood flow conditions. Other diseases associated with calcification may be managed through rectal or oral chelation.
Combining the chelation therapy with antibiotics has proven fruitful in treating diseases associated with nanobacteria. Although tetracycline remains the most effective antibiotic, individuals depicting hypersensitivity to the drug can take sulfa medicines and possibly some other antibiotics. It is possible that natural substitutes to prescription antibiotics may be used such as berberine, oil of oregano, colloidal silver, and other natural remedies. However, much research is needed in exploring the natural alternatives to affirm their efficacy.
As Rona (2005), reports, suggestions from Dr. Roberts, an authority in dealing with nanobacteria, indicate that “a combination of oral EDTA with supportive antioxidants plus an EDTA rectal suppository and 500 mg of tetracycline all given once daily before bedtime is the most effective way of ridding the body of both the pathological calcium deposits and the nanobacteria.” Further investigations into the procedure show that blood levels of EDTA remain high 24 hours a day when applying this practice. On the other hand, intravenous EDTA levels drop down to zero in a while after the intravenous drip is detached. Thus, emphasis is on high and stable levels of EDTA to ensure a rapid, more efficient chelation procedure.
Notably, the procedure for dealing with the nanobacteria is significantly different from the treatment of typical bacterial infections. In the case of typical bacterial infections, the body responds to the disease-causing bacteria by enhancing localized blood flow in the afflicted organ, a process known as inflammation. This is followed by the deployment of immune system cells to attack and terminate the bacteria. Similarly, the antibodies released by the immune system adhere to the bacteria and facilitate their destruction (Kajander, 2006). Following the administration of antibiotics, a series of reactions that disrupt the bacteria’s metabolic processes are initiated. Although strains that are antibiotic-resistant have emerged, vaccination helps to prevent many significant bacterial illnesses such as tetanus, Hemophilus influenza Type b (Hib), and whooping cough.
On the other hand, dealing with nanobacterial diseases requires a different approach altogether. The first stage of the anti-nanobacterial treatment involves weakening the hardened shells by introducing substances such as fulvic acid and liquid zeolites which breakdown the molecular bonds. This step is followed by incorporating EDTA and / or dimethyl sulfoxide as a way to inflict further damage on the calcified shells (Kajander, 2006). In the next step, tetracycline is introduced as it inhibits the apatite-binding protein formation, chelates calcium and hinders metalloproteinase. It also destroys the exposed nanobacteria, thus eliminating them from the body.
Considering its recent discovery, it is apparent that nanobacteria forms a sufficiently new scope of research in medicine. As such, our acknowledgement of nanobacterial infections can lead us to revisit our understanding chronic diseases and their symptoms’ presentations. Studying the nanobacteria serves to bridge the gap between the symptoms and the etiological causes in some chronic diseases, especially renal and cardio vascular diseases. Furthermore, due to the distinctive system of replication and metabolism, it means that only particular agents can upset and eliminate them. All in all, more research is needed to facilitate further understanding of the biological features of the nanobacteria.
References
Kajander, E. O. (2006). Nanobacteria–propagating calcifying nanoparticles. Letters in applied microbiology , 42(6), 549-552.
Rona, Z. P. (2005). The Nanobacteria Revolution .
