Anxiety disorder is an ailment of the nervous system that results from factors such genetic, metabolic and other biological elements. This disease is highly occurring to people suffering from psychiatric disorders. Usually, a person experiencing this condition goes through neuron-based alterations in body functions as well as mood changes. Some of the symptoms of anxiety disorder include excessive worry, sweating, hypervigilance, gastrointestinal changes and palpation that persists for a prolonged period ( Bilkei, Holsboer &Ströhle, 2010) . This analysis consequently, seeks to provide an elaborate discussion about the neurobiological basis of anxiety disorder as well as the types of drugs used to treat it.
From the evolutionary perspective, a human brain has both a newer and an ancient part of the cortex. In the former section also known as the prefrontal cortex, higher reasoning centers are contained which are responsible for thinking and planning ( Clement & Chapouthier, 2013). Further still, this part keeps in check the emotive reaction of a person. Emotional processing takes place in the latter section where a brain assembly known as the limbic system is enclosed. This structure plays a vital role in assimilating the affective, cognitive and sensory mechanisms of pain and processing information with regard to the inner bodily condition ( Clement & Chapouthier, 2013) .
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The limbic system also has one detrimental organ, hippocampus, which is involved in a central role of managing anxiety response by controlling over the hypothalamic stress-response system and managing the negative feedback relating to the hypothalamic-pituitary-adrenal axis ( Bilkei, Holsboer &Ströhle, 2010 ). Inherently, the growth of both the neurogenesis and hippocampus has a fundamental role in developing resilience regarding stress and anxiety disorders.
Amygdala is, however, the most crucial part in the limbic system that plays the primary duty of regulating emotions because it is essential in the development of emotionally noticeable external incitements and also the initiation of the suitable behavioral reaction ( Clement & Chapouthier, 2013) . Besides, it is involved in the manifestation of fear and violence and also helps in establishment and recovery of memories related to emotions and fear; thus, a victim with anxiety illness is seen to have an overly reactive amygdala.
For a person with anxiety disorders, benzodiazepines, tricyclic antidepressant, and barbiturates are used to effectively treat the anxiety symptoms such as psychomotor agitation ( Bilkei, Holsboer &Ströhle, 2010). In addition, serotonin-norepinephrine reuptake inhibitors (SRI) as well as selective serotonin reuptake inhibitors (SSRI) are usually recommended as the major drugs for first line treatment ( Bilkei, Holsboer &Ströhle, 2010) . Also, monoamine oxidase inhibitors such as paroxetine are administered to lower the signs of harm evasion.
Despite the increasing progression in the knowledge relating to neurobiological bases of anxiety disorders, many cases of anxiety still remains untreatable. With increasing research, however, it is apparent that as neuroimaging technologies continue to develop, better ways are bound to emerge in relation to how anxiety disorders are treated.
References
Bilkei-Gorzo, A., Holsboer, F., & Ströhle, A. (2010). Anxiety and anxiolytic drugs . Berlin [etc.: Springer
Clement, Y., & Chapouthier, G. (2013). Biological bases of anxiety . Neuroscience & Biobehavioral Reviews , 22 (5), 623-633.
